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A VIPoma is a neuroendocrine neoplasm secreting vasoactive intestinal peptide (VIP), usually presenting with severe watery secretory diarrhea, which can result in hypokalemia and metabolic acidosis and with flushes. Hypochlorhydria, stimulation of glycogenolysis, and hypercalcemia can be also found in VIPoma patients.
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Here, we describe a subpopulation of superficial cortical multipolar interneurons expressing vasoactive intestinal peptide (VIP) with high spine densities on their dendrites located in layer (L).
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VIP cells, one of the major inhibitory cell types in neocortex 5, 6, are commonly found in superficial layers 7. They preferentially inhibit somatostatin positive (SST) cells that mediate.
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VIP gene transcripts are mainly found in neurons or neuron-related cells. VIP gene expression is regulated by neuronal and endocrine signals that contribute to its developmental control. VIP exerts its function via receptor-mediated systems, activating signal transduction pathways, including cAMP.
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Vasoactive intestinal peptide (VIP) interneurons in sensory cortex modulate sensory responses based on global exploratory behavior and arousal state, but their function during non-exploratory, goal-directed behavior is not well understood.
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Inhibitory neurons which express vasoactive intestinal polypeptide, VIPs, are a small subset of the mammalian cortex but in importance live up to their acronym. New research shows that these critical control knobs of cortical activity are specifically activated by actions taken when rewards are anticipated rather than consummated. Main text
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2200184 DOI: 10.1016/0165-6147(90)90253-5 Abstract VIP-containing cells in the neocortex are intrinsic neurons of the bipolar type, which release VIP through mechanisms that involve Ca2+ and lipoxygenase metabolites.
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The VIP and L. casei ATCC 393 co-cultured cells were first administered with 1 mL of 1×10 8 CFU/mL of L. casei ATCC 393 culture supernatant for 3 h, then changed to 1 mL of FBS-free DMEM containing 0.1 µM VIP and culured for 12 h. Afterwards, the cell culture supernatants were collected, and the concentration of β-hexosaminidase, tryptase.
Pathway of VIP signal transduction and inflammatoryresponse regulation
VIP cells in the visual cortex activate differently to novel stimuli compared to familiar stimuli and, in the auditory cortex, to novel sounds and shocks during fear conditioning [22, 29].
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Vasoactive intestinal peptide (VIP) is a neuropeptide belonging to the VIP/secretin/glucagon family of peptides that can be released from both neurons and immune cells. From: Brain, Behavior, and Immunity, 2009 About this page VIP Sandrine Passemard,. Vincent Lelièvre, in Handbook of Biologically Active Peptides (Second Edition), 2013
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This supports the idea that VIP cells contribute to the difference in coherence between matched and non-matched surrounds. Importantly, the regression slopes for coherence were significantly different from those for power (Figure 4 K, p = 0.004, n = 9, Wilcoxon signed rank test). Download : Download high-res image (827KB)
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Using multielectrode recording and optogenetic manipulations, we show that VIP neurons provide coordinated daily waves of GABAergic input to target cells across the paraventricular hypothalamus.
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VIP (Vasoactive Intestinal Peptide) is a Protein Coding gene. Diseases associated with VIP include Vipoma and Vasomotor Rhinitis.Among its related pathways are GPCR downstream signalling and Presynaptic function of Kainate receptors.Gene Ontology (GO) annotations related to this gene include signaling receptor binding and neuropeptide hormone activity.
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Vasoactive intestinal peptide, also known as vasoactive intestinal polypeptide or VIP, is a peptide hormone that is vasoactive in the intestine. VIP is a peptide of 28 amino acid residues that belongs to a glucagon/secretin superfamily, the ligand of class II G protein-coupled receptors. [5]
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Inhibitory Neurons: Vip Cells Hit the Brake on Inhibition Recent studies on vasoactive intestinal peptide-expressing inhibitory neurons in the barrel and auditory cortices of the mouse brain have shown that they form a disinhibitory circuitry that affects the excitability of pyramidal neurons. Carsten K. Pfeffer